MARÍA BERROCAL CARRILLO
PROFESOR/A SUSTITUTO/A (TIEMPO COMPLETO)
Departamento de BIOQUÍMICA Y BIOLOGÍA MOLECULAR Y GENÉTICA
Área de BIOQUÍMICA Y BIOLOGÍA MOLECULAR
FACULTAD DE CIENCIAS
- Scopus: 36875549800
- Índice H: 15
Grupo de investigación
BIOMARCADORES DE NEURODEGENERACIÓN EN CEREBRO
Instituto de investigación
Instituto Universitario de Biomarcadores de Patologías Moleculares
Asignaturas
Consulta toda la información investigadora de MARÍA BERROCAL CARRILLO en el Portal de Investigación.
Tesis:
Publicaciones:
- 2025: Neurotoxic amyloid β‐peptide and tau produce cytokine‐like effects on PMCA in glioblastoma cell lines, enhancing its activity and isoforms expression
- 2023: Store-Operated Calcium Entry Inhibition and Plasma Membrane Calcium Pump Upregulation Contribute to the Maintenance of Resting Cytosolic Calcium Concentration in A1-like Astrocytes
- 2023: The Plasma Membrane Ca2+-ATPase, a Molecular Target for Tau-induced Cytosolic Calcium Dysregulation
- 2022: Inhibition of SERCA and PMCA Ca2+-ATPase activities by polyoxotungstates
- 2021: Gold compounds inhibit the ca2+-atpase activity of brain pmca and human neuroblastoma sh-sy5y cells and decrease cell viability
- 2021: Sorcin activates the brain pmca and blocks the inhibitory effects of molecular markers of alzheimer’s disease on the pump activity
- 2019: Correction to: STIM1 deficiency is linked to Alzheimer’s disease and triggers cell death in SH-SY5Y cells by upregulation of L-type voltage-operated Ca2+ entry (Journal of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-y)
- 2019: Methylene Blue Blocks and Reverses the Inhibitory Effect of Tau on PMCA Function
- 2018: Methylene blue activates the PMCA activity and cross-interacts with amyloid β-peptide, blocking Aβ-mediated PMCA inhibition
- 2018: STIM1 deficiency is linked to Alzheimer’s disease and triggers cell death in SH-SY5Y cells by upregulation of L-type voltage-operated Ca2+ entry
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